Bacterial Endocarditis and Mural Thrombi

نویسندگان

  • B. WALLACH
  • MACELLIS GLASS
  • LESLIE LUKASH
  • A. ANGRIST
چکیده

EMBOLIC phenomena occur with such frequency in the course of bacterial endocarditis as to constitute a cardinal diagnostic feature of this disease. However, it has been noted that the resultant infarcts are less often septic than would be expected if the emboli originated from the infected valvular vegetations.' A previous study of this apparent paradox showed that it is the nonbacterial thrombotic vegetation that is a frequent source of these emboli in patients with rheumatic heart disease with or without superimposed bacterial endocarditis.2 An incidental feature of this analysis, further substantiating the above findings, was the relative rarity of mural thrombi as the origin of such bland emboli in the cases of bacterial endocarditis. Another report also indicated the relative infrequency of severe mitral stenosis with bacterial endocarditis as well as the greater rarity of auricular fibrillation in patients with bacterial endocarditis.3 The role of each of these factors in the formation of mural thrombi in the left auricle has been discussed previously.4 A perusal of the recent literature discloses few references dealing specifically with this subject. Garvin5 in a study of mural thrombi in the heart found only 1 instance of mural thrombus in 30 cases of subacute bacterial endocarditis (3.3 per cent) and none in 13

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تاریخ انتشار 2005